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Hiraishi Masaya

Faculty of Pharmaceutical Sciences Molecular Pharmaceutical Sciences Molecular and Cellular Biological SciencesAssistant Professor

Researcher basic information

■ Degree
  • PhD (Veterinary Medicine), Hokkaido University, Mar. 2026
■ URL
researchmap URLホームページURL■ Various IDs
J-Global ID

Career

■ Career
Career
  • Apr. 2026 - Present
    Hokkaido University, Faculty of Pharmaceutical Sciences, 助教
  • Apr. 2023 - Mar. 2026
    日本学術振興会 特別研究員 DC1
  • Apr. 2020 - Mar. 2022
    JA全農, 家畜衛生研究所
Educational Background
  • Apr. 2022 - Mar. 2026, Hokkaido University, School of Veterinary Medicine
  • Jul. 2024 - Feb. 2025, The University of Melbourne, School of Agriculture, Food and Ecosystem Sciences, Study Abroad Research Student, Australia
  • Apr. 2014 - Mar. 2020, Hokkaido University, Faculty of Veterinary Medicine

Research activity information

■ Awards
  • Apr. 2023, 日本獣医解剖学会, 獣医解剖分科会奨励賞(一般部門)
    全身性自己免疫疾患モデルマウスにみられる頭部リンパ組織の形態変化
  • Oct. 2022, Asian Association of Veterinary Anatomists, Best Oral Presentation Award
    Altered morphologies of tear-secreting tissues and cornea in a mouse model of autoimmune disease.
  • Sep. 2021, 関東・東京合同畜獣医師大会・三学会, 産業動物部門大会長賞
    国内で検出された豚サーコウイルス2型に対する市販ワクチンの有効性確認
  • Sep. 2019, 日本獣医解剖学会, 獣医解剖分科会奨励賞(学部学生部門)
    自己免疫疾患モデルマウスにみられる涙液産生組織の形態学的変化
■ Papers
  • Morphological abnormalities in the ureter of type XVII collagen-deficient mice.
    Amy Suzui; Takashi Namba; Masaya Hiraishi; Sao Oe; Shunnosuke Kira; Yuki Otani; Rui Zhong; Ken Natsuga; Osamu Ichii
    Scientific reports, 16, 1, 27 Apr. 2026, [International Magazine]
    English, Scientific journal, Non-fibrillar type XVII collagen (COL17A1) is a hemidesmosomal component of the epidermis and a key skin-disease molecule. We previously reported that COL17A1 is expressed in the urothelium of multiple species at low constitutive or in inducible levels. To clarify its function in the ureter, Col17a1-knockout (KO) mice were examined from 2 to 12 weeks of age. After 4 weeks, Col17a1-KO mice showed shorter ureteral length, smaller cross-sectional area, and disorganized urothelial layers compared with wild-type littermates, accompanied by systemic growth retardation. At 4 and/or 12 weeks, Col17a1-KO urothelium exhibited a higher epithelial area ratio, loss of polarity, intraluminal cells, lumen narrowing, and indistinct mucosal folds. Abnormalities were evident as early as 2 weeks, particularly in umbrella cells, which showed altered apical surfaces, hypertrophy, smaller vesicles, and shortened interdigitations. RNA-seq of 2-week-old Col17a1-KO ureters revealed upregulation of immune-related genes and downregulation of epithelial development genes. Uroplakin 2, a key urothelial molecule, was reduced in Col17a1-KO ureters at 1 day and 2 weeks but not at 12 weeks. Its transcription factor, forkhead box protein A1, was persistently reduced, with markedly lower protein expression in umbrella cells. These findings indicate that urothelial COL17A1 is essential for proper urothelial differentiation during postnatal development.
  • Dominance of conjunctiva-associated lymphoid tissue in the feline ocular immune system, with identification of lacrimal duct-associated lymphoid tissue.
    Masaya Hiraishi; Takashi Namba; Yuki Otani; Shunnosuke Kira; Osamu Ichii
    The Journal of veterinary medical science, 88, 1, 82, 89, 16 Jan. 2026, [Lead author], [Domestic magazines]
    English, Scientific journal, Mucosa-associated lymphoid tissue, which plays a crucial role in both immunological protection and inflammation at mucosal sites, shows interspecies variation in its distribution. In the ocular region, lacrimal duct-associated lymphoid tissue (LDALT) has rarely been investigated in animals other than humans or rodents. Although conjunctiva-associated lymphoid tissue (CALT) has been reported in various species, its cell composition remains unclear. Herein, we aimed to elucidate the morphology and development of the feline ocular immune system by examining the lacrimal tract and its associated tissues in adolescent (7- to 8-month-old) and fetal cats. The feline lacrimal tract, composed of the lacrimal canaliculi, lacrimal sac, and nasolacrimal duct, was visualized using CT. Histologically, a small lymphocyte aggregate, identified as LDALT, was observed exclusively along the lacrimal canaliculus in one of the four adolescent cats. In contrast, extensive development of the CALT was observed in tissues surrounding the openings of the lacrimal tract, particularly on the cornea-facing side of the third eyelid. CALT is characterized by CD20+ B cell aggregates surrounded by CD3+ T cells, including a small subset coexpressing CD20 and CD3. Neither LDALT nor CALT was observed in late gestational fetuses. In conclusion, CALT appears to contribute more significantly than LDALT to feline ocular immunity. Furthermore, given the pro-inflammatory nature of both CALT and CD3+CD20+ cells, our findings suggest that CALT is involved in the pathology of feline conjunctivitis.
  • Age- and sex-dependent morphological alterations of rectal mucosa-associated lymphoid tissue in chickens (Gallus gallus domesticus).
    Md Zahir Uddin Rubel; Takashi Namba; Masaya Hiraishi; Sao Oe; Osamu Ichii
    Poultry science, 104, 11, 105846, 105846, Nov. 2025, [International Magazine]
    English, Scientific journal, The gut mucosal immune system plays a pivotal role in maintaining intestinal homeostasis, defending against pathogens, and shaping microbiota composition in both mammals and birds that develop mucosa-associated lymphoid tissue (MALT). Here, we characterized the formation and maturation of rectal-MALT (RMALT) in chickens. Given that this maturation critically affects immune competence and health, this study aimed to characterize the developmental dynamics of chicken RMALT, considering sex differences. We investigated RMALT in both sexes across three age groups (1 day, 1 month, and 1 year) using integrated histomorphological and functional analyses. Histologically, RMALT in both sexes increased in size and progressed from diffuse lymphoid aggregations to solitary follicles with age. Immunohistochemistry showed that RMALT is composed of CD3⁺ T cells, Bu-1⁺ B cells, MHCII⁺ antigen-presenting cells, and KUL01⁺ monocyte/macrophage. Notably, 1-year-old females possessed larger RMALT sizes than their male counterparts. Both sexes showed >90% PCNA⁺ proliferating cells in B-cell regions, but females had significantly more proliferating cells in T-cell areas, suggesting enhanced cellular activity. Furthermore, we examined the antigen uptake pathway in the rectal epithelium by focusing on goblet and microfold cell-like cells. In both sexes of 1-year-old chickens, the follicle-associated epithelium showed fewer goblet cells than the non-follicle-associated epithelium regions. Moreover, Jacalin- CSF1R⁺ microfold cell-like cells in the follicle-associated epithelium significantly increased with aging. Thus, the age-related progression of RMALT suggests a pivotal role in maintaining local immunological conditions in the rectum, particularly in females. These findings provide foundational insights into avian mucosal immunity, which could inform strategies for improving poultry health management.
  • Sex-related differences in the morphology of rectal mucosa-associated lymphoid tissues in C57BL/6NCrSlc mice.
    Md Zahir Uddin Rubel; Md Abdul Masum; Takashi Namba; Masaya Hiraishi; Yasuhiro Kon; Osamu Ichii
    Histology and histopathology, 40, 8, 1195, 1208, Aug. 2025, [International Magazine]
    English, Scientific journal, Sex hormones regulate gut function and mucosal immunity; however, their specific effects on the mucosa-associated lymphoid tissue (MALT) in the rectum of mammals remain unclear. Here, we aimed to investigate the influence of sex on MALT in the rectum of mammals by focusing on the rectal mucosa-associated lymphoid tissues (RMALTs) of C57BL/6NCrSIc mice. Histological analysis revealed that RMALTs were predominantly located in the lamina propria and submucosa of the rectal mucosa, with a significant sex-related difference in the distance from the anorectal junction to the first appearance of the RMALT. Despite similar RMALT numbers, females exhibited significantly larger RMALTs than males. Immunostaining revealed the presence of various immune cells, including T cells, B cells, macrophages, proliferative immune cells, lymphatic vessels, and high endothelial venules (HEVs), in RMALTs. Compared with males, females showed elevated T cell, helper T cell, and cytotoxic T-cell gene expression levels, along with high percentages of specific T-cell subsets. The factors influencing RMALT development, such as the presence of HEVs, C-X-C motif chemokine ligand 13 expression, and RMALT-containing cell proliferation, were also explored. Overall, this study revealed the detailed attributes of RMALTs, their immune cell composition, and their determinants in male and female mice, providing insights into the sex-specific characteristics of the rectal mucosal immune system.
  • Altered morphology of mucosa-associated lymphoid tissues and epithelium in the nasal cavity and lacrimal apparatus in autoimmune disease-prone MRL/MpJ-Faslpr/lpr mice
    Masaya Hiraishi; Takashi Namba; Teppei Nakamura; Md. Zahir Uddin Rubel; Yasuhiro Kon; Osamu Ichii
    Cell and Tissue Research, 400, 3, 331, 345, Springer Science and Business Media LLC, 25 Mar. 2025, [Lead author], [International Magazine]
    English, Scientific journal, The mucosal epithelium and the mucosa-associated lymphoid tissues (MALTs) protect the mucosa, such as eye and nose, from constant exposure to foreign antigens. As autoimmune disorders can target both epithelium and MALTs, we morphologically investigated the head of MRL/MpJ-Faslpr/lpr, an autoimmune disease-prone mouse, to discuss the pathological crosstalk among autoimmune disorders, mucosal epithelium, and MALTs. Compared to healthy control MRL/MpJ mice, MRL/MpJ-Faslpr/lpr mice had more lymphoid tissues that were diffusely localized beneath the mucosa epithelium in their lacrimal tracts and nasal cavity. Particularly, lacrimal duct- and nasopharynx-associated lymphoid tissues (LDALT, NALT) were identified as the major MALTs in the mouse head. In the nasal mucosa, MRL/MpJ-Faslpr/lpr mice exhibited larger NALT, more frequent non-ciliated epithelial cells, and more goblet cells than in MRL/MpJ mice. NALT in MRL/MpJ-Faslpr/lpr mice contained more proliferating cells than in MRL/MpJ mice. Moreover, LDALT in some MRL/MpJ-Faslpr/lpr mice developed by being directly connected to the bone marrow surrounding the nasolacrimal duct. These data suggested systemic autoimmune disorders could alter the mucosal immunity of the head by affecting both mucosal epithelium and MALTs. These findings are crucial for understanding the pathology of the head mucosal immunity.
  • Application of chlorous acid water for disinfection of surgical site in dairy cows.
    Osamu Ichii; Teppei Nakamura; Masaya Hiraishi; Takashi Namba; Md Zahir Uddin Rubel; Takuya Umeyama; Megumi Asai
    Frontiers in veterinary science, 12, 1444674, 1444674, 2025, [International Magazine]
    English, Scientific journal, Disinfection is crucial for preventing surgical site infections. Recently, the effectiveness of sanitizers using chlorous acid (HClO2) under conditions rich in organic matter has been reported, and chlorous acid water (CAW) has been approved as a food additive. This study evaluated the potential of CAW as a new presurgical disinfectant for cattle. The experiments were performed on the paralumbar fossa of cattle in Sapporo during March (winter to spring) and August (summer). Colony-forming units (CFUs) of standard plate count bacteria (SPCB), Enterococcus faecalis (EF), Pseudomonas aeruginosa, Escherichia coli, and Staphylococcus spp. (SP) were analyzed as indicators of bacterial load. SPCB and SP were abundantly detected, exceeding 6 log10 CFU/100 g on clipped hair and 6 log10 CFU/100 cm2 on the skin immediately after clipping, with no significant seasonal differences. The bacterial load on the skin was evaluated at three time points: after clipping, cleansing, and disinfection. Clipping and cleansing with liquid soap were common procedures, following this, either the standard disinfection protocol using 7.5% iodine scrub for 1 min, 10% povidone-iodine for 5 min, and 70% alcohol for 5 min (SPA), or a modified protocol using CAW with contact times of 15, 10, or 5 min (CAW15, CAW10, CAW5) were performed separately. The cleansing procedure significantly reduced the SPCB, EF, and SP on the skin after clipping, and all disinfection methods significantly decreased the SP after cleansing. Draping significantly enhanced the disinfection efficiency of the SPA, CAW10, and CAW5 protocols. The CAW procedure did not alter skin histology in the paralumbar fossa or udder compared to 10% povidone-iodine or 70% alcohol. Our data suggest that the disinfection method using CAW is useful and comparable to routine disinfection methods and might reduce the time required for presurgical disinfection in farm fields.
  • Systemic autoimmune abnormalities alter the morphology of mucosa-associated lymphoid tissues in the rectum of MRL/MpJ-Faslpr/lpr mice.
    Md Zahir Uddin Rubel; Osamu Ichii; Takashi Namba; Md Abdul Masum; Tsolmon Chuluunbaatar; Masaya Hiraishi; Teppei Nakamura; Yasuhiro Kon
    Experimental animals, 73, 3, 270, 285, 09 Jul. 2024, [Domestic magazines]
    English, Scientific journal, Systemic autoimmune diseases (ADs) might affect the morphology and function of gut-associated lymphoid tissue (LTs) indirectly; however, their exact relationship remains unclear. Therefore, we investigated mouse LTs in the anorectal canal and morphologically compared them between MRL/MpJ-Fas+/+ and MRL/MpJ-Faslpr/lpr mice. LT aggregations, also known as rectal mucosa-associated lymphoid tissues (RMALTs), were exclusively seen in the lamina propria and submucosa of the rectum. The mean size and number of the LT aggregations both significantly increased in MRL/MpJ-Faslpr/lpr mice compared to those in MRL/MpJ-Fas+/+ mice. The distance from the anorectal junction to the first LT aggregate was significantly shorter in MRL/MpJ-Faslpr/lpr mice than that in MRL/MpJ-Fas+/+ mice. Immunostaining revealed that the RMALTs included CD3+, CD4+, and CD8+ T cells; B220+ B cells; IBA1+ macrophages; Ki67+ proliferative cells; and PNAd+ high-endothelial venules (HEVs). The numbers of macrophages, proliferative cells, CD4+ T cells, CD8+ T cells, and HEVs were significantly increased in MRL/MpJ-Faslpr/lpr mice compared to those in MRL/MpJ mice. Furthermore, the gene expression levels of chemokines (Cxcl9 and Cxcl13) and their corresponding receptors (Cxcr3 and Cxcr5) were significantly higher in MRL/MpJ-Faslpr/lpr mice than those in MRL/MpJ-Fas+/+ mice. Although the morphology of rectal epithelium was comparable between the strains, M cell number was significantly higher in MRL/MpJ-Faslpr/lpr mice than in MRL/MpJ-Fas+/+ mice. Thus, ADs could alter RMALT morphology, and quantitative changes in T-cell subsets, proliferative cells, macrophages, HEVs, chemokine expression, and M cells could affect their cell composition and development.
  • Roles of tumor necrosis factor-like ligand 1A in γδT-cell activation and psoriasis pathogenesis.
    Shangyi Wang; Mina Kozai; Masaya Hiraishi; Md Zahir Uddin Rubel; Osamu Ichii; Mutsumi Inaba; Kazuhiro Matsuo; Kensuke Takada
    Frontiers in immunology, 15, 1340467, 1340467, 2024, [International Magazine]
    English, Scientific journal, BACKGROUND: Interleukin (IL)-17-producing γδT (γδT17) cells mediate inflammatory responses in barrier tissues. Dysregulated γδT17 cell activation can lead to the overproduction of IL-17 and IL-22 and the development of inflammatory diseases, including psoriasis. IL-23 and IL-1β are known to synergistically activate γδT17 cells, but the regulatory mechanisms of γδT17 cells have not been fully elucidated. This study aimed to reveal the contribution of the inflammatory cytokine tumor necrosis factor-like ligand 1A (TL1A) to γδT17 cell activation and psoriasis development. METHODS: Anti-TL1A antibody was injected into an imiquimod (IMQ)-induced murine psoriasis model. TL1A receptor expression was analyzed in splenic and dermal γδT cells. γδT cells were tested for cytokine production in vitro and in vivo under stimulation with IL-23, IL-1β, and TL1A. TL1A was applied to a psoriasis model induced by intradermal IL-23 injection. Mice deficient in γδT cells were intradermally injected with IL-23 plus TL1A to verify the contribution of TL1A-dependent γδT-cell activation to psoriasis development. RESULTS: Neutralization of TL1A attenuated γδT17 cell activation in IMQ-treated skin. TL1A induced cytokine production by splenic γδT17 cells in synergy with IL-23. Dermal γδT17 cells constitutively expressed a TL1A receptor at high levels and vigorously produced IL-22 upon intradermal IL-23 and TL1A injection but not IL-23 alone. TL1A exacerbated the dermal symptoms induced by IL-23 injection in wild-type but not in γδT cell-deficient mice. CONCLUSION: These findings suggest a novel regulatory mechanism of γδT cells through TL1A and its involvement in psoriasis pathogenesis as a possible therapeutic target.
  • Phenotypes of streptozotocin-induced gestational diabetes mellitus in mice.
    Narumi Takahashi; Osamu Ichii; Masaya Hiraishi; Takashi Namba; Yuki Otani; Teppei Nakamura; Yasuhiro Kon
    PloS one, 19, 4, e0302041, 2024, [International Magazine]
    English, Scientific journal, Gestational diabetes mellitus (GDM) in human patients disrupts glucose metabolism post-pregnancy, affecting fetal development. Although obesity and genetic factors increase GDM risk, a lack of suitable models impedes a comprehensive understanding of its pathology. To address this, we administered streptozotocin (STZ, 75 mg/kg) to C57BL/6N mice for two days before pregnancy, establishing a convenient GDM model. Pregnant mice exposed to STZ (STZ-pregnant) were compared with STZ-injected virgin mice (STZ-virgin), citrate buffer-injected virgin mice (CB-virgin), and pregnant mice injected with citrate buffer (CB-pregnant). STZ-pregnant non-obese mice exhibited elevated blood glucose levels on gestational day 15.5 and impaired glucose tolerance. They also showed fewer normal fetuses compared to CB-pregnant mice. Additionally, STZ-pregnant mice had the highest plasma C-peptide levels, with decreased pancreatic islets or increased alpha cells compared to CB-pregnant mice. Kidneys isolated from STZ-pregnant mice did not display histological alterations or changes in gene expression for the principal glucose transporters (GLUT2 and SGLT2) and renal injury-associated markers. Notably, STZ-pregnant mice displayed decreased gene expression of insulin-receiving molecules (ISNR and IGFR1), indicating heightened insulin resistance. Liver histology in STZ-pregnant mice remained unchanged except for a pregnancy-related increase in lipid droplets within hepatocytes. Furthermore, the duodenum of STZ-pregnant mice exhibited increased gene expression of ligand-degradable IGFR2 and decreased expression of GLUT5 and GLUT12 (fructose and glucose transporters, respectively) compared to STZ-virgin mice. Thus, STZ-pregnant mice displayed GDM-like symptoms, including fetal abnormalities, while organs adapted to impaired glucose metabolism by altering glucose transport and insulin reception without histopathological changes. STZ-pregnant mice offer a novel model for studying mild onset non-obese GDM and species-specific differences in GDM features between humans and animals.
  • Species-specific histological characterizations of renal tubules and collecting ducts in the kidneys of cats and dogs.
    Shunnosuke Kira; Takashi Namba; Masaya Hiraishi; Teppei Nakamura; Yuki Otani; Yasuhiro Kon; Osamu Ichii
    PloS one, 19, 7, e0306479, 2024, [International Magazine]
    English, Scientific journal, The histomorphological features of normal kidneys in cats and dogs have been revealed despite the high susceptibility of cats to tubulointerstitial damage. Herein, the histological characteristics of the two species were compared. Cytoplasmic lipid droplets (LDs) were abundant in the proximal convoluted tubules (PCTs) of cats aged 23-27 months but scarce in dogs aged 24-27 months. LDs were rarely observed in the distal tubules (DTs) and collecting ducts (CDs) of either species, as visualized by the expression of Tamm-Horsfall protein 1, calbindin-D28K, and aquaporin 2. The occupational area ratio of proximal tubules (PTs) in the renal cortex was higher, but that of DTs or CDs was significantly lower in adult cats than in dogs. Single PT epithelial cells were larger, but PCT, DT, and CD lumens were significantly narrower in adult cats than in dogs. Unlike adults, young cats at 6 months exhibited significantly abundant cytoplasmic LDs in proximal straight tubules, indicating lipid metabolism-related development. Histochemistry of the 21 lectins also revealed variations in glycosylation across different renal tubules and CDs in both species. Sodium-glucose cotransporter 2 was expressed only in PTs, excluding the proximal straight tubules with few LDs in adult cats or the PCTs of young cats and adult dogs. These findings are crucial for understanding species-specific characteristics of renal histomorphology and pathogenesis.
  • Structural Features of Connective Tissue Formed around Resin Implants Subcutaneously Embedded in Dairy Cows.
    Yuka Katayama; Osamu Ichii; Teppei Nakamura; Keita Yanase; Masaya Hiraishi; Takashi Namba; Yuki Otani; Teppei Ikeda; Erika Tsuji; Natsuko Tsuzuki; Ken Kobayashi; Yasuhiro Kon; Takanori Nishimura
    Animals : an open access journal from MDPI, 13, 23, 29 Nov. 2023, [International Magazine]
    English, Scientific journal, Foreign body reactions (FBRs) are inadvertently observed in invading or artificially embedded materials, triggering inflammation and subsequent fibrotic processes to occur in situ. Here, we assessed the spatiotemporal formation of connective tissue around implanted materials to establish a technique using connective tissue formed by FBRs as xenografts. An acrylic resin implant, comprising a columnar inner rod and a tubular outer cylinder (OC) with several slits, was embedded in adult dairy cows. Tissues formed in the inner rod and OC groups were histologically analyzed at weeks 2, 4, 8, and 12. Edematous tissues with non-collagenous fibers formed for 2 weeks and showed increased cellularity after 4 weeks. The weight, thickness, amounts of total protein, collagen, DNA, and quantitative scores of α-smooth muscle actin-positive myofibroblasts or elastic fibers notably increased after 8 weeks, with condensed collagen fibers showing orientation. Inflammatory cells were primarily localized in tissues close to the OC, and their numbers increased, with the count of CD204+ cells peaking at 8 weeks and declining at 12 weeks. The count of Ki67+ proliferating cells slightly increased in tissues close to the OC; however, the number and lumen of CD31+ vessels increased. These results may help understand FBR-related tissue remodeling.
  • Histopathological changes in tear-secreting tissues and cornea in a mouse model of autoimmune disease.
    Masaya Hiraishi; Md Abdul Masum; Takashi Namba; Yuki Otani; Yaser Ha Elewa; Osamu Ichii; Yasuhiro Kon
    Experimental biology and medicine (Maywood, N.J.), 245, 12, 999, 1008, Jun. 2020, [Lead author], [International Magazine]
    English, Scientific journal, Cornea, an outermost layer of mammalian eye, is protected by tear film and abnormalities of tear film causes dry eye. Dry eye injures the cornea which results lower vision in patients. Several factors cause dry eye, including altered systemic conditions, environment, and immunological abnormality of the patient in autoimmune disease like Sjögren's syndrome (SS). However, the detailed pathology of autoimmune abnormality-mediated dry eye is unclear. Here we demonstrated that systemic autoimmune abnormality in BXSB-Yaa mice was associated with histological changes in the exocrine glands and cornea of the eyes. We also showed that BXSB-Yaa mice developed mild or early stage dry eye-like disease and explain the existence of a compensatory mechanism associated with the dysfunction of these tissues. Thus, BXSB-Yaa could be a model for SS-like disease-associated dry eye and these data would contribute to the understanding of the pathogenesis of autoimmune-related dry eye disease.
■ Other Activities and Achievements
■ Affiliated academic society
  • 日本免疫学会
  • 日本解剖学会
  • 日本獣医学会
■ Research Themes
  • ヒツジの眼局所免疫が備える暑熱耐性メカニズムの解明
    若手研究人材育成事業(若手研究人材・ネットワーク育成補助金(タレント補助金))
    Jul. 2026 - Mar. 2027
    ノーステック財団, 北海道大学, Principal investigator
  • 動物の眼局所免疫が示す暑熱応答についての基礎研究 -暑さに強い畜産動物の作出を見据えて-
    令和8年度研究助成
    Jun. 2026 - Mar. 2027
    公益社団法人 栗林育英学術財団, 北海道大学, Principal investigator
  • IgA腎症モデルマウスから解き明かす動物の頭部免疫―腎臓軸とその病態
    科学研究費助成事業
    25 Apr. 2023 - 31 Mar. 2026
    平石 真也
    日本学術振興会, 特別研究員奨励費, 北海道大学, 23KJ0070
  • 暑さに強い羊 -Dorper種がもつ暑熱耐性メカニズムの解明-
    くら基金
    Jul. 2024 - Feb. 2025
    平石 真也; Surinder Singh Chauhan
    公益財団法人公益推進協会, メルボルン大学
  • 反芻動物のメタン排出抑制がその健康に与える影響の解明
    若手研究者海外挑戦プログラム
    Jul. 2024 - Feb. 2025
    平石 真也
    日本学術振興会, メルボルン大学
  • 動物の頭部粘膜を保護する腺-免疫連関とその異常
    DX博士人材フェローシップ
    Apr. 2022 - Mar. 2023
    平石 真也
    北海道大学
  • IgA 腎症モデルマウスの解析からみる動物の頭部免疫 ― 腎臓連関
    卓越大学院科学研究費
    Nov. 2022 - Feb. 2023
    平石 真也
    北海道大学
■ Academic and Social Contribution Activities/Other
Others
  • Jun. 2026 - Present
    北海道大学男子ラクロス部 顧問
  • Sep. 2024 - Present
    肉眼動物解剖技術者(日本獣医解剖学会)
  • Apr. 2022 - Mar. 2026
    北海道大学One Healthフロンティア卓越大学院 修了生
  • Feb. 2024
    第15回HOPEミーティング参加者(日本学術振興会)
  • Apr. 2022 - Mar. 2023
    北海道大学DX博士人材フェローシップ